Familial Idiopathic Basal Ganglia Calcification (Fahr's syndrome): Initial Clinical Neuropsychiatric Presentation without Corresponding Neurological Deficit

Malte Christian Claussen *

Department of Psychiatry and Psychotherapy, University Hospital Zurich, University of Zurich, Zurich, Switzerland

Katayun Hassanpour

Department of Psychiatry and Psychotherapy, University Hospital Zurich, University of Zurich, Zurich, Switzerland

Josef Jenewein

Department of Psychiatry and Psychotherapy, University Hospital Zurich, University of Zurich, Zurich, Switzerland

Soenke Boettger

Department of Psychiatry and Psychotherapy, University Hospital Zurich, University of Zurich, Zurich, Switzerland

*Author to whom correspondence should be addressed.


Abstract

Familial idiopathic basal ganglia calcification (FIBGC) or Fahr's syndrome is a rare disorder with various clinical presentations which can mimic - in particular - psychiatric illness. The following case is characterized by the typical basal ganglia calcifications and presentation of neuropsychiatric symptoms indicating the first clinical presentation in the absence of a neurological deficit. As previously reported, the extent of calcification did not predict neurological impairment, however, predicted severe psychosis.

Keywords: Basal ganglia, calcification, familial idiopathic basal ganglia calcification (FIBGC), Fahr's syndrome, neurological deficits, psychosis, extra-pyramidal symptoms


How to Cite

Christian Claussen, Malte, Katayun Hassanpour, Josef Jenewein, and Soenke Boettger. 2017. “Familial Idiopathic Basal Ganglia Calcification (Fahr’s syndrome): Initial Clinical Neuropsychiatric Presentation Without Corresponding Neurological Deficit”. International Neuropsychiatric Disease Journal 9 (3):1-5. https://doi.org/10.9734/INDJ/2017/34524.

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